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Senescent Cells Are Essentially Cells Research Paper

When DNA is damaged, cells can react through cell cycle checkpoints which allows repair to begin before further division can occur. There is also the prokaryotic SOS response which changes gene expression in bacteria as a result of DNA damage. This response is regulated by the production of certain proteins. Moreover, eukaryotic cells also react to DNA damage through producing proteins that begin the process of DNA repair. 8. Mice are often the favored mammalian in the testing of aging interventions. This is often because of the fact that there are many mice easily available for testing but also the fact that "generation time is short" (Yuan et al. 2011). Essentially, the aging process and testing in interventions can be done on a much shorter time scale then with tests using monkeys.

9. Enzymes may change the transcription patterns of a cell by adding chemical groups to histone proteins. This process increases the sensitivity to DNase I digestion.

10. Old stem and progenitor cells in a young systemic environment can be rejuvenated. Heterochronic parabiosis can be restored through the activation of Notch signaling and a "regenerative capacity of aged satellite cells" (Conboy et al. 2005).

References

Books. W.R. Clark. (2009). Human genetic diseases that mimic the aging process. Progeria? The...

Web. http://www.progeriaproject.com/progeria/mimic.htm
Conboy, I.M., Conboy, M.J., Wagers, a.J., Girma, E.R., Weissman, I.L., & Rando, T.A. (2005). Rejuvenation of aged progenitor cells by exposure to a young systemic environment. Nature, 17(433), 760-764.

Conti, Matteo. (2008). The Selfish Cell: An Evolutionary Defeat. Springer Publishing.

McClintock, D., Ratner, D., Lokuge, M. (2007). The mutant form of Lamin a that causes Hutchinson-Gilford Progeria is a biomarker of cellular aging in human skin. PLOS One, 2(2). Web. http://www.plosone.org/article/info:doi/10.1371/journal.pone.0001269

Science Daily. (2011). New clues about aging: Genetic splicing mechanism triggers both premature aging syndrome and normal cellular aging. Science News. Web. http://www.sciencedaily.com/releases/2011/06/110613121956.htm

Wade, Nicholas. (2011). Senescent cells hasten aging but can be purged mouse study suggests. New York Times. Web. http://www.nytimes.com/2011/11/03/science/senescent-cells-hasten-aging-but-can-be-purged-mouse-study-suggests.html

Yuan, ROng, Peters, Luanne L., & Paigen, Beverly. (2011). Mice as a mammalian model for research on the genetics of aging. U.S. National Library of Medicine. Web. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3074346/

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References

Books. W.R. Clark. (2009). Human genetic diseases that mimic the aging process. Progeria? The Progeria Project Foundation. Web. http://www.progeriaproject.com/progeria/mimic.htm

Conboy, I.M., Conboy, M.J., Wagers, a.J., Girma, E.R., Weissman, I.L., & Rando, T.A. (2005). Rejuvenation of aged progenitor cells by exposure to a young systemic environment. Nature, 17(433), 760-764.

Conti, Matteo. (2008). The Selfish Cell: An Evolutionary Defeat. Springer Publishing.

McClintock, D., Ratner, D., Lokuge, M. (2007). The mutant form of Lamin a that causes Hutchinson-Gilford Progeria is a biomarker of cellular aging in human skin. PLOS One, 2(2). Web. http://www.plosone.org/article/info:doi/10.1371/journal.pone.0001269
Science Daily. (2011). New clues about aging: Genetic splicing mechanism triggers both premature aging syndrome and normal cellular aging. Science News. Web. http://www.sciencedaily.com/releases/2011/06/110613121956.htm
Wade, Nicholas. (2011). Senescent cells hasten aging but can be purged mouse study suggests. New York Times. Web. http://www.nytimes.com/2011/11/03/science/senescent-cells-hasten-aging-but-can-be-purged-mouse-study-suggests.html
Yuan, ROng, Peters, Luanne L., & Paigen, Beverly. (2011). Mice as a mammalian model for research on the genetics of aging. U.S. National Library of Medicine. Web. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3074346/
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