Of a total of 21 cases which were examined, this procedure resulted in a slight complication in only one case, where the digit had to be removed surgically due to failure of removal by suture ligation. The only real side effect of this simple treatment is that there was found to be a small bump remaining at the site in 43% of cases.
Conclusion
Although there has been some work into the genetic basis of polydactyly, advances in examination of the human genome may create a better understanding of the condition in the future. A large amount of the work done so far has been focused on animal models, and there is still room for further work to discover the genetic basis of the different types of polydactyly in humans.
References
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Boeing, M., Cassia F. Paiva, L., Lima Garcias, G., Graca Martino Roth, M. & Santos, I.S. (2001). Epidemiologia das polidactilias: Um estudo de casos e controles na populacao de Pelotas-RS. Journal de Pediatria, 77(2); d.o.i.: 10.1590/S0021-7557200100017.
Borisch, N., Stunitz, B. & Blauth, W. (1995). Case histories surgical treatment of polydactyl of the little toe involving proximal and middle phalanx. Orthopedics and Traumatology, 4(4): 246-253.
Gurnett, C.A., Bowcock, a.M., Dietz, F.R., Morcuende, J.A., Murray, J.C. & Dobbs, M.B. (2007). Two novel point mutations in the long-range SHH enhancer in three families with triphalangeal thumb and preaxial polydactyly. American Journal of Medical Genetics, 143(1): 27-32.
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