41). Groups like the ALA fund research on various forms of COPD every year, so it seems certain that some kind of additional treatments and preventions may be discovered in the future. Since emphysema is such a prevalent disease, continued research must be completed to help ease the suffering of millions of Americans, and save lives, too.
In conclusion, emphysema is a serious and deadly disease that can be prevented in most people by simply avoiding cigarettes. The disease can be treated, but once diagnosed, it cannot be cured. It affects millions of Americans, and studies show it will continue to affect Americans who smoke, even if they have quit smoking years before. While there have been many studies done on the disease, researchers still do not know how to recondition the lungs once they are affected. Emphysema is serious, and more studies need to be done to help determine…...

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References

Author not Available. (Nov. 2004). Emphysema. Retrieved from the American Lung Association Web site:   April 2005.http://www.lungusa.org/site/pp.asp?c=dvLUK9O0E&b=35043#whatis20 

Berardelli, P. (1997, October 27). Of mice and men. Insight on the News, 13, 41.

Honish, R.L. (1982). Chest and lung diseases. In Over 55: A handbook on health, Duncan, T.G. (Ed.) (pp. 81-100). Philadelphia: Franklin Institute Press.

Lewis, C. (1999, March). Every breath you take. FDA Consumer, 33, 9.

Understanding Bronchiectasis Outline

Introduction

Brief overview of bronchiectasis
Importance of awareness and understanding of the condition


Understanding Bronchiectasis
Definition and explanation of bronchiectasis
The anatomy and function of the bronchi
How and why bronchiectasis affects the lungs
Detailed explanation of causes leading to bronchiectasis
Exploration of risk factors including genetic predispositions, environmental factors, and associated conditions
Common symptoms associated with bronchiectasis
Diagnostic processes and tests used to identify bronchiectasis
Current treatment options available for bronchiectasis
Long-term management strategies for living with bronchiectasis
Preventive measures to reduce the risk of developing bronchiectasis
Future research directions and potential advancements in treatment
Summary of key points discussed
The importance of early diagnosis and effective management
Encouragement for readers to seek medical advice if they experience symptoms
Causes and Risk Factors
Symptoms and Diagnosis
Treatment and Management
Prevention and Future Outlook
Conclusion


Introduction

Bronchiectasis is a chronic lung condition that has remained in the shadows of more commonly discussed respiratory diseases like asthma and chronic obstructive pulmonary disease (COPD). However, its impact on patients' lives is no less significant,…...

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Resources

Pasteur, M.C., Bilton, D., & Hill, A.T. (2010). British Thoracic Society guideline for non-CF bronchiectasis. Thorax, 65(Suppl 1), i1-i58.

Flume, P.A., Mogayzel, P.J., Robinson, K.A., Goss, C.H., Rosenblatt, R.L., Kuhn, R.J., & Marshall, B.C. (2007). Cystic fibrosis pulmonary guidelines: chronic medications for maintenance of lung health. American Journal of Respiratory and Critical Care Medicine, 176(10), 957-969.

McShane, P.J., Naureckas, E.T., Tino, G., & Strek, M.E. (2013). Non-cystic fibrosis bronchiectasis. American Journal of Respiratory and Critical Care Medicine, 188(6), 647-656.

Chalmers, J.D., Aliberti, S., & Polverino, E. (2015). The EMBARC European Bronchiectasis Registry: protocol for an international observational study. ERJ Open Research, 1(1), 00081-2015.

Introduction BPD or bronchopulmonary dysplasia represents a chronic ailment of the lungs, largely occurring among premature babies requiring oxygen therapy and mechanical ventilation for treating severe respiratory distress. It may also develop among immature babies displaying symptoms of early lung-related illness or babies born at their term but requiring rigorous ventilator therapy to treat serious lung ailments. In the last four decades, prenatal steroid administration, surfactant therapy, better nutrition, novel ventilator strategies and other modern treatments have led to significant progress in the medical course and results of premature babies suffering from respiratory distress syndrome (RDS). But in spite of the above advancements, BPD prevalence, on the whole, has remained the same over the last ten years (Gien & Kinsella, 2011).
Bronchopulmonary dysplasia pathophysiology
Babies displaying the greatest likelihood of BPD diagnosis are born at a time when their lungs haven’t yet transitioned to the saccular stage from the canalicular stage. Considering the…...

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References

Abman, S. H., Collaco, J. M., Shepherd, E. G., Keszler, M., Cuevas-Guaman, M., Welty, S. E., ... & Kirpalani, H. (2017). Interdisciplinary care of children with severe bronchopulmonary dysplasia. The Journal of pediatrics, 181, 12-28.

Collins, J. J., Tibboel, D., de Kleer, I. M., Reiss, I. K., & Rottier, R. J. (2017). The future of bronchopulmonary dysplasia: emerging pathophysiological concepts and potential new avenues of treatment. Frontiers in medicine, 4, 61.

Gien, J., & Kinsella, J. P. (2011). Pathogenesis and Treatment of Bronchopulmonary Dysplasia. Current Opinion in Pediatrics, 23(3), 305–313.

Patel, V. & Cherian S. (2016). Management of Bronchopulmonary Dysplasia / Chronic lung disease. Retrieved 17 May 2018 from http://www.cardiffnicu.com/Portal/Respiratory/Chronic%20lung%20disease%20guideline.pdf

Tropea, K., & Christou, H. (2012). Current pharmacologic approaches for prevention and treatment of bronchopulmonary dysplasia. International journal of pediatrics, 2012.

http://doi.org/10.1097/MOP.0b013e328346577f

Some of the major objectives of the strategy include lessening regional alveolar distension, atelectasis, oxygen mediated injury, diaphragm injury, and inflammation. The other approaches that can be used to lessen the injury include using high frequency oscillatory ventilation, positioning, neuromascular blocking agents, fluid therapy, and immunomodulation.
Conclusion:

Ventilator Induced Lung Injury is one of the common illnesses that occur among patients with acute lung injury. This disease is mainly attributed to the use of mechanical ventilator to save these patients though there are other risk factors that contribute to the injury.

eferences:

Dreyfuss, D. & Saumon, G. (1998, January 1). Ventilator-induced Lung Injury -- Lessons from Experimental Studies. American Journal of espiratory and Critical Care Medicine, 157(1), 294-323. etrieved from http://ajrccm.atsjournals.org/content/157/1/294.long

Feng et. al. (2011, July 19). Pediatric Acute espiratory Distress Syndrome Treatment and Management. etrieved September 26, 2012, from http://emedicine.medscape.com/article/803573-treatment

Galvin, S. & Granton, J. (2011, March 3). educing Ventilator Lung Injury. etrieved September

26,…...

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References:

Dreyfuss, D. & Saumon, G. (1998, January 1). Ventilator-induced Lung Injury -- Lessons from Experimental Studies. American Journal of Respiratory and Critical Care Medicine, 157(1), 294-323. Retrieved from  http://ajrccm.atsjournals.org/content/157/1/294.long 

Feng et. al. (2011, July 19). Pediatric Acute Respiratory Distress Syndrome Treatment and Management. Retrieved September 26, 2012, from  http://emedicine.medscape.com/article/803573-treatment 

Galvin, S. & Granton, J. (2011, March 3). Reducing Ventilator Lung Injury. Retrieved September

26, 2012, from  http://respiratory-care-sleep-medicine.advanceweb.com/Features/Articles/Reducing-Ventilator-Induced-Lung-Injury.aspx

he most common progressive chronic lung situations that would require the rehabilitation include interstitial lung disease, chest wall disease, bronchiectasis, and pre and post thoracic surgery. he fourth category of patients to offered pulmonary rehabilitation is those with recent exacerbation of COPD requiring hospitalization, without the anticipated recovery path, and whose functional baseline has changed significantly ("Service Specification," 2012).
he Problem:

Chronic lung diseases have developed to become one of the most common respiratory illnesses across the country. As the diseases have become one of the major reasons for hospitalizations of patients, they usually affect individuals at the age of 35 years and above despite of the fact that these individuals are usually not diagnosed until they are 50 years and above. he growth and rapid increase of chronic diseases is attributed to the tendency of many people with the disease not to get medical assistance. While the conditions continue to…...

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The most common progressive respiratory disease is the Chronic Obstructive Pulmonary Disease (COPD), which is a name that is also used to refer to a collection of lung diseases ("Introduction," 2012). The other common chronic lung diseases include emphysema, chronic bronchitis, chest wall disease, interstitial lung disease, chronic asthma, and obstructive airways disease. These chronic lung diseases usually involve a combination of three major factors i.e. airway diseases, lung circulation diseases, and lung tissue diseases. As the name suggests, the airway diseases affect the airways or tubes that transport oxygen and other gases to and from the lungs. In contrast, the lung tissues diseases affect the lung tissue structure through inflammation or scarring of the tissue. Lung circulation diseases affect the blood vessels in the lungs through scarring, clotting, or inflammation of these vessels. As a result, these diseases contribute to difficulties of the lungs to receive oxygen and release carbon dioxide. The severity of chronic lung diseases is basically dependent on the combination of these three conditions. However, most of the chronic lung diseases are attributed to the narrowing or blockage of the airways. For instance, emphysema, COPD, and chronic bronchitis are conditions that inhibit the ability of the tubes or airways to carry oxygen and other gases to and from the lungs. Generally, patients with chronic lung diseases such as COPD always have difficulties breathing because of airflow obstruction or narrowing of the airways.

As previously mentioned, these diseases account for a huge number of hospital emergency room visits and hospitalizations, although they are largely preventable. Despite being preventable, these diseases have become some of the major causes of death and key factors in the ever-increasing huge human and economic burden because of the tendency of patients to ignore their symptoms ('Emergency Department Support Fund Application," n.d). The major symptoms of these diseases include rising breathlessness, frequent chest infections, and constant cough with phlegm ("Introduction," 2012). The tendency to ignore these symptoms contributes to the many incidents of recidivism to acute care facilities.

The main cause of chronic lung diseases, especially chronic obstructive pulmonary disease is smoking. An individual enhances the risk of developing these diseases when he/she smokes more and for long periods of time. Smoking results in scarring that increases the risk of chronic lung diseases, through irritating or inflaming the lungs. The inflammation in turn results in permanent lung changes over many years. During this period, the walls of the tubes or airways thicken as more mucus is generated. In addition to making the lungs lose their normal elasticity, the damage or harm to the delicate walls of the air sacs in the lungs results in the development of emphysema. Moreover, the smaller airways or tubes become narrowed or scarred. The combination of these permanent changes to the lungs contributes to symptoms of cough, breathlessness, and phlegm linked to chronic obstructive pulmonary

Pulmonary Sarcoidosis
Sarcoidosis is a sometimes-lethal disease affecting primarily the lungs and thoracic lymphatic system, and its hallmark feature is noncaseating granulomas in multiple tissues and organs (Hoang and Nguyen, 2010, p. 36; American Thoracic Society, 1999, p. 736). Over 90% of all sufferers have pulmonary involvement, but granulomas are frequently found in other organs and tissues, including the skin, eyes, liver, spleen, parotid glands, central nervous system, muscles, bones, and genitourinary tract (Hoang and Nguyen, 2010, p. 36). When death does result, it is typically due to pulmonary fibrosis. What follows is a review of pulmonary sarcoidosis from a clinical perspective.

Causes and isk Factors

The cause of sarcoidosis is unknown, but research into the nature of the resulting granulomas suggests immune dysregulation in genetically susceptible individuals is the primary causative factor (American Thoracic Society, 1999, p. 738-740). The genetic contribution appears to be significant, as evidenced by an ethnic, gender, and…...

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References

American Thoracic Society. (1999). Statement on Sarcoidosis. American Journal of Respiratory Critical Care Medicine, 160, 736-755.

American Lung Association. (2010). State of lung disease in diverse communities 2010. Lung.org. Retrieved 5 Mar. 2012 from  http://www.lung.org/assets/documents/publications/lung-disease-data/solddc_2010.pdf .

Baughman, Robert P., Lower, Elyse E., and du Bois, Roland M. (2003). Sarcoidosis. Lancet, 361, 1111-1118.

Drent, Marjolein, De Vries, Jolanda, Lenters, Merinke, Lamers, Rob J. s., Rothkranz-Kos, Snjezana, Wouters, Emiel F.M. et al. (2003). Sarcoidosis: Assessment of disease severity using HRCT. European Radiology, 13, 2462-2471.

Population of the City of Atlantis on March 30, 2003 = 183,000
of new active cases of TB occurring between January 1 and June 30, 2003 = 52

of active TB cases according to the city register on June 30, 2003 = 238

The incidence rate of active cases of TB for the 6-month period was: [ONE POINT]

per 100,000 population per 100,000 population per 100,000 population per 100,000 population

130 per 100,000 population

183,000 is 183% of 100,000, so the rate per 100,000 would be 52 divided by 1.84...or 28.

The prevalence of active TB as of June 30, 2003, was: [ONE POINT]

14 per 100,000 population

130 per 100,000 population

144 per 100,000 population

264 per 100,000 population

e. none of the above

B -- using the same math as above...except it's 238 / 1.84 -- 129.3

3. Which of the following is an advantage of active surveillance? [ONE POINT]

a. requires less project staff

b. is relatively inexpensive to employ

c. more accurate…...

Inside the alveoli, oxygen moves across the paper-thin walls to the capillaries, the tiny blood vessels, and then into the blood, where it is picked up by chemicals, haemoglobin, in the red blood cells ready to be carried throughout the body (ay pp). At the same time, waste products from the body, in the form of carbon dioxide, come out of the capillaries back into the alveoli, ready to be breathed out (ay pp). Freshly oxygenated blood is carried from the lungs to the left side of the heart which pumps blood around the body through the arteries, and once the oxygen is used up, the blood returns, through the veins, to the right side of the heart (ay pp). From there, it is pumped to the lungs so that the carbon dioxide can be removed and more oxygen taken in (ay pp).
Every day, some 10,000 liters of air…...

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Works Cited

The Way Our Lungs Work. British Lung Foundation.  http://www.lunguk.org/the-way-our-lungs-work.asp

Empyema Clinical Manifestation
Empyema: Lung Sounds and other Clinical Manifestations

Over the past decade, empyema has consistently been recognized as an acute, potentially life-threatening respiratory disease. A large number of studies have been conducted over the last years that address the unique symptomatology of empyema and the clinical implications of these symptoms.

The biggest diagnostic challenge of empyema is that patient often present with symptoms very similar and difficult to distinguish from an uncomplicated pneumonia. A patient typically shows symptoms such as fever and chills, excessive sweating, malaise, cough, dyspnea, pleuritic chest pain and unintentional weight loss (Sahn, 2007). These symptoms individually do not warrant a diagnosis of empyema. They do, however, require precautionary follow-up testing that should include a pleural fluid aspiration. The presence of pus -- an opaque, whitish-yellow viscous fluid consisting of serum coagulation proteins, cellular debris and fibrin deposition -- aspirated from the pleural space is a direct indication…...

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References:

1. Walker W, Wheeler R, Legg J. (2011). Update on the causes, investigation and management of empyema in childhood. Archives of Disease in Childhood, 96, 5, 482-488.

2. Sahn SA. (2007). Diagnosis and management of parapneumonic effusions and empyema. Clinical Infectious Diseases, 45, 11, 1480-1486.

3. Heffner JE, Klein JS, Hampson C. (2010). Diagnostic utility and clinical application of imaging for pleural space infections. Chest, 137, 2, 467-479.

4. Froudarakis ME. (2008). Diagnostic Work-Up of Pleural Effusions. Respiration, 75, 4-13.

Physiological Effects of Hodgkin's Disease
In this paper I shall give an overview of Hodgkin's disease while focusing on its physiological effects. Specifically, the paper consists of an overview of the disease, describes how the disease affects the body cells and tissues, and how the treatment attacks the disease and affects the body, besides reviewing the treatments available.

Hodgkin's disease is one of the two (and less severe) types of cancer of the lymphatic system; the other type being non-Hodgkin's lymphoma. The disease is named after the British physician, Thomas Hodgkin, who first discovered the condition in 1832. Hodgkin's disease commonly occurs in young adults (between the ages of 15 to 35) and in older people (over 50-year-olds. However, about 10%-15% of cases have been diagnosed in children below 16 years of age. Statistics also show that more men than women are afflicted by it. ("What are the Key Statistics ... "…...

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References

'Autologous Bone Marrow Stem Cell Transplantation" (2004). American Cancer Society. Retrieved on September 28, 2004 from   'Chemotherapy." (2004). American Cancer Society. Retrieved on September 28, 2004 from  http://www.cancer.org/docroot/CRI/content/CRI_2_4_4X_Chemotherapy_20.asp?rnav=cri  'Do We Know What Causes Hodgkin's Disease?" (2004). American Cancer Society. Retrieved on September 28, 2004 from  http://www.cancer.org/docroot/CRI/content/CRI_2_4_2X_Do_we_know_what_causes_Hodgkins_disease_20.asp?rnav=cri http://www.cancer.org/docroot/CRI/content/CRI_2_4_4X_Autologous_Bone_Marrow_Stem_Cell_Transplantation_and_Peripheral_Blood_Stem_Cell_Transplantation_20.asp?rnav=cri 

"Hodgkin's Disease." (2000) The Columbia Encyclopedia, Sixth Edition. Columbia University Press: New York.

'Hodgkin's disease: Overview" (2004) Oncology Channel Retrieved on September 28, 2004 from / 'How is Hodgkin's Lymphoma and the Non-Hodgkin's Lymphomas Different?" (2004) Lymphoma Information Network. Retrieved on September 28, 2004 from  http://www.lymphomainfo.net/lymphoma/comparison.html http://www.oncologychannel.com/hodgkins

'How Is Hodgkin's Disease Treated?" (2004). American Cancer Society. Retrieved on September 28, 2004 from   'The Lymphatic System." (2004) CancerBACUP. Retrieved on September 28, 2004 from  http://www.cancerbacup.org.uk/Cancertype/LymphomaHodgkins/General/Thelymphaticsystemhttp://www.cancer.org/docroot/CRI/content/CRI_2_4_4X_How_Is_Hodgkins_Disease_Treated_20.asp?rnav=cri 

Creatine treatment started at 6, 8, and 10 weeks of age, analogous to early, middle, and late stages of human HD, significantly extended survival at both the 6- and 8-week starting points. Significantly improved motor performance was present in both the 6- and 8-week treatment paradigms, while reduced body weight loss was only observed in creatine-supplemented R6/2 mice started at 6 weeks." (Dedeoglu, et al., 2003) Specifically it is stated that the "...Neuropathological sequelae of gross brain and neuronal atrophy and huntington aggregates were delayed in creatine-treated R6/2 mice started at 6 weeks. We show significantly reduced brain levels of both creatine and ATP in R6/2 mice, consistent with a bioenergetic defect. Oral creatine supplementation significantly increased brain concentrations of creatine and ATP to wild-type control levels, exerting a neuroprotective effect. These findings have important therapeutic implications, suggesting that creatine therapy initiated after diagnosis may provide significant clinical benefits…...

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Bibliography

NINDS Huntington's Disease Information Page (2009) National Institute of Neurological Disorders and Stroke. Online available at  http://www.ninds.nih.gov/disorders/huntington/huntington.htm 

Hamilton, J.M., et al. (2004) Rate and Correlates of Weight Change in Huntington's Disease. Journal of Neurology Neuroscience and Psychiatry 2004; 75:209-212. BMJ Publishing Group Ltd. Online available at  http://jnnp.bmj.com/cgi/content/abstract/75/2/209 

Gaba, Ann M. et al. (2005) Energy Balance in Early-Stage Huntington Disease. American Journal of Clinical Nutrition, Vol. 81, No. 6. June 2005.

Djousse, L. (2002) Weight Loss in Early Stage of Huntington's Disease. Journal of Neurology 2002. Nov. 12:59(9): 1325-30.

" This drug has already won approval for use in Europe and the United tates. tudies conducted show that the drug "targets the tumor to control in four areas: in the site where hypersecretion starts, in GH secretion, IGF-1 and in the symptoms associated with the disease (Unknown, 2004)." While the drug has been approved, there are still contraindications to taking it such as a patient who has an irregular or slow heart rate, or blood sugar levels which are either too high or too low.
Occurrence

Although gigantism begins prior to puberty, the "majority of giants eventually demonstrate features of acromegaly, of which the mean age for the onset is within the 3rd decade of life. Even a congenital onset of GH excess has been suggested by linear growth acceleration occurring within the first few months of life in young children with documented gigantism (http://jcem.endojournals.org/cgi/content/full/84/12/4379)." Although there is no accurate number…...

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Simmons, Kirt E. (01 October, 1999). "Growth Hormone and Craniofacial Changes: Preliminary Data From Studies in Turner's Syndrome." Pediatrics.

Skatssoon, Judy. (21 May, 2002). "NSW: New discovery could lead to cure for dwaftism, gigantism." AAP General News (Australia).

Unknown. (01 June, 2004). "Novartis Sandostatin LAR approved in Japan for Acromegaly. Worldwide Biotech.

HEALTH
COLON CANCE

Colon cancer is defined as a cancer that begins in the rectum or colon. The cancers are referred to as colon or rectal cancer with reference to their orientating point. Colon and rectal cancers share different features and ultimate diagnosis procedures. The disorders are discussed collectively in this paper including their treatment approach.

In case cancer forms within polyps, they eventually start growing into the colon or rectal walls. While cancer cells attach to the wall, they continue growing within blood and lymph vessels. The lymph vessels are relatively thin, and tiny channels are carrying away fluid and waste (Acton, 2013). They occasionally drain into subsequent lymph nodes that are bean-shaped structures and contain immune cells to helping fighting the infections. Immediately the cancer cells start spreading to lymph or blood vessels, they travel to the lymph nodes and other distant body parts like the liver. The process of cancer…...

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References

Acton, Q.A., (2013). Colon Cancer: New Insights for the Healthcare Professional: 2013. New York: ScholarlyEditions.

Peterson, E., (2014). The Colon Cancer. New York: Sea Salt.

Young, A., Hobbs, R., & Kerr, D., (2011). ABC of Colorectal Cancer. New York: John Wiley & Sons.

Pressure on the superior vena cava may produce SVC syndrome, a swelling of the head and arms. SVC syndrome involving the brain can be fatal and must be treated immediately. But enlarged lymphatic tissue in the chest cavity generally tends to displace -- rather than press upon or encase -- adjacent structures. Therefore, compromised breathing and SVC syndrome are relatively uncommon signs of lymphoma. (Hodgkin's Disease, 1998-2008)
Effects on Bone Marrow

Night sweats, fevers or anemia (a low red-blood-cell count), fevers may indicate Hodgkin's disease has spread to an individual's bone marrow. In these scenarios, a physician may order bone marrow aspiration and biopsy. In biopsy, medical staff uses a large needle to remove a narrow, cylindrical piece of the patient's bone. In another option, medical staff performs an aspiration, a process utilizing a needle to remove small bits of bone marrow. Generally, in both instances, to help determine cancer spread,…...

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Works Cited

Atlas of the Body: The Lymphatic System." (1999). American Medical Association. 2 June 2008  http://www.medem.com/medlb/article_detaillb.cfm?article_ID=ZZZG0S6CGJC&sub_at=518 .

Carson-DeWitt, Rosalyn S; Alic, Margaret. "Hodgkin's Disease," Gale Encyclopedia of Cancer, January 1, 2002. 2 June 2008  http://www.highbeam.com/doc/1G2-3405200219.html .

Detailed Guide: Hodgkin Disease What Is Hodgkin Disease? American Cancer Society. Revised: 08/30/2007. 2 June 2008  http://www.cancer.org/docroot/CRI/content/CRI_2_4_1x_What_Is_Hodgkin_Disease.sp?rnav=cri .

Hodgkin's Disease Signs and Symptoms. (1998-2008). 3 June 2008 http://www.oncologychannel.com/hodgkins/symptoms.shtml.

, 1998). It is hard to know where the boundary stops between psychological and physical illness, since the two are, often intimately combined with one affecting the other.
QOL, as De Vries and Drent (2008) point out is often confounded with state of physical health but actually it reflects one's emotional and psychological welfare. Nonetheless, the two are intimately related in that each affects the other.

Conducting a through review on the subject with key words involving 'Sarcoidosis and health status', Sarcoidosis and quality of life" or Sarcoidosis and fatigue" De Vries and Drent (2008) ended up with 15 studies that they considered relevant to their subject.

Counter-intuitively, they discovered that the greatest challenge on QOL as effected by was the patient's fatigue caused by the disease. Breathlessness, reduced exercise, and impaired working and physical activities were the most frequent reported hindrances. The instrument used was the World Health Organization Quality of Life…...

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References

American Thoracic Society (1999) Statement on Sarcoidosis, 736-749

The report provides a thorough overview of Sarcoidosis discussing new developments and demonstrating how much in the field remains enigmatic.

Bona, J. et al. (1998) Neurosarcoidosis as a Cause of Refractory Psychosis: A Complicated Case ReportAm J. Psychiatry 155:8, 1100-1107

The report describes Sarcoidosis and gives a case history as example.