Methylphenidate is part of a therapy regimen for the control of the symptoms of Attention Deficity Hyperactivity Disorder or ADHD in adults and children (Ogbru 2013, Medline Plus 2012). It stimulates the central nervous system similarly as amphetamines but more mildly. The effects of methylphenidate are also more noticeable on mental activities than in physical movements. It also is used in treating narcolepsy and Attention Deficit Disorder or ADD. Stimulants control these symptoms by changing the amount of natural substances in the brain responsible for the conditions. But both classes of stimulants are carefully used because of their potential abuse. They calm the patient, reduce their hyperactivity and increase attention span. FDA approved methylphenidate in 1955 (Ogbru, MedlinePlus).
Generic and Trade Names
Generic names -- methylphenidate, Methylphenidylacetate hydrochloride
Trade names -- Concerta, Metadate, Methylin, Ritalin, Adderall
Toxicity and Side Effects
It is to be used with cause on patients with relatives who suffer from Tourette's psychosis, emotional instability, major depression, glaucoma or motor tics (NIDA 2009, MedlinePlus 2012, Ogbru 2013). It must also be used cautiously on patients with a history of substance or alcohol abuse. Long-term abuse...
Mindful vs. traditional martial arts toward improved academic grades in children diagnosed with ADHD While medication and psychotherapy are the current best practice in treating attention deficit hyperactivity disorder (ADHD), their benefits and aim are too peripheral and topical -- neither resolving the neurological origin of deficits. Moreover, many are opposed to these treatments and there are few substantiated and readily accepted alternatives. The consequences of ADHD have a ripple effect --
When processed by a transglutaminase enzyme, it can interact with immunological cells and produce cytotoxic inflammation. In autism, it is believed that peptides from gluten and casein cross the intestinal microvillus barrier and enter the blood stream. They also cross the blood-brain barrier. In the brain, certain amino acid sequences of these peptides compete with natural peptides, which bind to opioid receptors. These receptors are G-protein receptors in cell
Michelson explains that buspirone could manipulate certain serotonin receptors in an attempt to ameliorate the overload of serotonin, and that amantadine was thought to increase dopamine activity. As such, either might theoretically help with SSRI-related sexual dysfunction. However, when the double-blind test was performed, it found that the success of treatment was roughly the same regardless of whether these pills were taken or a placebo was used. One significant
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