Fatigue Management in Aviation
Many documented incidents can be linked to pilot fatigue. A case in kind occurred on August 18, 1993, where a Connie Kalitta DC-8 crashed whilst completing its 1/4-mile base leg. The flight crew had flown for 9 hours and been on duty for 18 hours, accordingly disrupting their circadian rhythm and experiencing sleep loss (National Transportation Safety Board, 1993).
Showing how fatigue was determined to be a contributing safety factor in the event
That the accident was, to a great extent, contributory to sleep loss was confirmed by Jim Danaher, chief of the NTSB's Operational Factors Division at the November 1995 Fatigue Symposium near Washington, D.C.:
The company had intended for the crew to ferry the airplane back to Atlanta after the airplane was offloaded in Guantanamo Bay. This would have resulted in a total duty time of 24 hours and 12 hours of flight time…."(National Transportation Safety Board, 1993).
The NTSB accident report also noted the cause of the accident as likely attributable to "The impaired judgment, decision-making, and flying abilities of the captain and flight crew due to the affects of fatigue" (ibid).
The report proceeds to list other causes, but fatigue is indicated as the primary factor. This, notes Brandon Printup (2000), is rare since fatigue is usually listed as an 'additional cause' with some other factor presented as primary contribution. Here, lack of sleep assumes foremost position whilst the Additional Factors section, in this instance, only displayed criticism on the FAA for inadequate flight and duty time regulations which factors, incidentally, conspired to produce existence of fatigue.
III. How contemporary fatigue science supports my argument that the significant causal factors of the event were fatigue impairment of the flight crew
Scientific research on sleep presents something of a paradox since, whilst on the one hand, it indicates that sleep deprivation is a condition that can be rectified and relatively insignificant, on the other hand, evidence seems to indicate that sleep deprivation may cause major, even fatal, harm. It is for this reason that sleep deprivation is not taken seriously enough, but, in reality, the extent of the harm caused by sleep deprivation depends on a variety of factors including the amount of sleep lost and on other aspects of the individual's circumstances, environment, and life (Weiten, 184). Negative effects of lack of sleep are most likely to occur when the individual has to deal with stress in his or her job or personal situation, when this stress is long lasting, or when individuals are faced with long-lasting, monotonous tasks. Negative effects of sleep deprivation are also likely to occur when individuals restrict their sleep to 6 hours or less for repetitive nights in a row (Weiten, 184). Then again, some individuals are more sensitive to sleep restriction than are others. Approximately, 20% of adults are routinely sleep deprived (Dumer & Dinges, 2005).
a. Neurobiology of the sleep-wake regulation cycle
The negative effects of sleep deprivation are due to the fact that the neurobiology of the regular sleep-wake regulation cycle is disrupted. There are four stage of sleep with the first called REM (rapid eye movement) where the mind is still partially active. This is succeeded by the next three stages where sleep as we know it occurs, and then again a brief interlude of REM during which stage dreams occur. This cycle occurs various times during the night, and the whole is moderated and modulated by a biological clock (otherwise known as circadian rhyme or changes) that is located in the suprachiasmatic nuclei that is in the hypothalamus. The biological clock does not only deal with sleepiness but affects all neurobehavioral variables with its circadian rhythm. This includes cognitive performance. What this means is that on a regular sleep-wake pattern, an individual can usually think (therefore function) in a consistently coherent, stable manner. Deprivation of sleep interrupts this circadian rhythm and may elevate homeostatic pressure of this clock to the extent that waking neurocognitive functions are weakened so that they function poorly even during the period of peak circadian wakefulness (Dumer...
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