Mostly proteins and platelets combine to form blood clotting in order to heal wounds inside the arteries. However, the accumulation of fatty protein and platelet deposits on the artery walls can sometimes cause the arteries to become narrow and hardened. This makes the artery more prone to damage and if it ruptures, the blood clotting occurs and thus, the artery becomes infected by arterial thrombosis.
Impact of patient age on the path physiology of CVI and DVT
The age factor is critical towards understanding how a disease might impact the sick person and how the person's body would respond to the various treatments applied on it. As mentioned earlier, these disorders are more common in the adult population. In the case of CVI, the vein walls weaken as the age of the person increases. This is due to the reason that the internal system of the person has become old, weak and slow. Consequently, the muscle layers in the veins of an infected area easily breakdown and the growth of CVI is quicker as compared to in a person of young age. Similarly, the DVT is also dependent upon the age of the patient.
Treatment of CVI and DVT...
Those who are the vulnerable will have specific genes they inherit from their relatives, who were impacted by the condition. This increases their chances of having similar complications when they become older. (Kolata, 2010) Congestive Heart Failure and Hypertension Congestive heart failure and hypertension are interconnected with each other. According to a study conducted by the University of Texas, they found that there are 660 thousand new cases reported each year.
Comparison of pathophysiology of CVI and DVT The pathogenesis of CVI is not completely understood; however, it's based on both venous reflux and obstruction; or an amalgamation of the two. Though venous reflux is actually based on a number of mechanisms, the key elements are venous valve ineffectiveness, vessel wall swelling, hemodynamic elements and additionally venous hypertension. These systems could be further exasperated by dysfunctional pumping devices (vascular and/or muscle pump),
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