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Crimes Against Children - Shaken Term Paper

However, the case studies ignore the discrimination of the incidence of the problem by race or socio economic classification. (Showers, 1992) Generally, the brain and the blood vessels of the babies are considered to be highly vulnerable to the whiplash injuries as a result of their anatomic structures. Besides the head of the baby constitutes about 10% of the weight of the body which is only 2% among the adults. At infancy the muscles at neck are very weak in comparison to that of any other stages in the life. The weak neck muscles along with the weak head control fails to resist the force exerted by the whiplash. Moreover the tender cranial joints and open fontanelles of the babies are conducive to worsen the slitting and trimming effects of the forces exerted. As a result of this a slight whiplash movement stretches the brain and the blood vessels instantly. The linking veins of the brain are not strong enough to resist the slitting stresses and results in hemorrhages.

The unmyelinated brain of the baby contributes towards making it more tender and are conducive to slitting of the brain and the bridging blood vessels making the brain more vulnerable to the trimming influences. The ventricles and subarachnoid spaces of the babies are filled with comparatively more amounts of cerebrospinal fluid- CSF, that allows the brain to dislocate more quickly and more intensively at the time of violent shaking that progressively increases stretching and the following vessel trimmings. The common form of intracranial injury related to the SBS is the subdural hematoma and accepted as the most common cause of death among the shaking infants. The subdural bleeding is observed to have occurred as a result of the slitting of the bridging veins interlacing between the brains to the sagittal sinus. This may also give rise to subarachnoid hemorrhages that will cause the spinal fluid taps to be bloody. (Smith, 2003)

In some cases the executor clutches the baby so tightly that causes the return of the thorax and venous from blood vessels in the head to decline following an enhanced intracranial pressure and cerebral edema. Cerebral edema may constitute the only finding in diagnoses process sometimes coupled with subarachnoid hemorrhage. The subdural bleeding often is venous rather than arterial and as a result is slower. This may prolongs the appearance of the neurological distortion from 24 to 48 hours. As a result of the significant bleeding the subarachnoid hemorrhages being arterial are considered fatal.

One significant observation in respect of the SBS is intraocular bleeding. The retinal hemorrhages are considered to be a common symptom of the SBS that purports to diagnose for intracranial injury and are not associated with other visible external wounds. According to Kivlin the retinal hemorrhages are taken to be the most common ocular observation in SBS. These are found in about 50% to 100% of the children. An instantaneous enhancement in the intracranial pressure is remitted to the eyes through the optic nerve covering resulting in the enhanced intraocular pressure. The associated intracranial and retinal damages result in long-term and enduring visual deficiency in case of the 30% to 80% of the victimized infants. The retinal hemorrhages present with the children of below 4 years are required to be diagnosed in terms of SBS. The supplementary clinical observations are associated with traction abrasions of the periosteum of the long bones without fractures or staining. In about 25% of SBS incidents the Radiographs of the long bones disclose about the old and new fractures of the infants. (Smith, 2003)

Moreover, multiple metaphyseal fractures are evident in cases of proximal humerus, distal ulna and radius, distal femur, and proximal and distal tibia and fibula and sometimes are left unacknowledged. The presence of the neurogenic pulmonary edema-NPE in SBS cases are observed by Rubin, McMillan, Helfaer and Christian. Irregular symptoms occur in case of the patho-physiology of NPE. According to some analysts quick increase in intracranial pressure encourages a catecholamine surge which is considered critical for the promotion of pulmonary edema. Such theory of NPE blast injury emphasizes that an aggressive increase in the intracranial pressure next to the central nervous system abuse give rise to excessive catecholamine release and a significant increase in the peripheral vascular resistance. Such an enhancement is followed by a retransmission of blood from the normal circulation to the lower resistance vascular bed of the lungs. Combined with the vasoconstriction of the pulmonary bed the effects are observed on a momentary propagation in pulmonary capillary wedge pressure encouraging the...

(Smith, 2003)
Child abuse refers to a state of atmosphere where the distortions in family interaction results in direct damage to the baby. Three elements are considered significant in the occurrence of distortions in the relationship in shaken baby syndrome. The behavioral problems with the parents and the hardship in social and stress management within the family contribute towards the physical abuse of the babies. The aggressive parents most often have an insensible attitude of role reversal demanding the child to be self nurturing and self protecting. The aggressive parents are identified with high level of stress in comparison to the non-aggressive parents. Even remaining the level of stress remaining constant the aggressive parents are more prone to resort to violence as a means of solving the problem in comparison to the non-abusive parents. The babies most of the time are involved in crying. (Spaide; Swengel; Scharre; Mein, 1990)

In an analysis made by Brazelton the infants of six weeks old are estimated to involve in crying for about two hours and forty five minutes per day on an average. In about 20 to 30% of infants between the six weeks to four months old, the colic and benign paroxysmal intestinal pain is inherent and irrational crying is very common. The aggressive parents who anticipates the baby to provide nurture and comfort feels dejected with the baby involved in such distressing crying. This gives the natural circumstances that lead to the momentum of the dejection and disappointment of the parent that naturally results in shaking of the baby. Normally inflicting injury to the babies are not the prime objective of the parents they simply demands a submissive or subservient child. The temporary dullness and drowsiness of the child as a result of shaking is expected by the parents and such consequences give rise to the strengthening of the parental behavior.

The detection of shaken baby syndrome is thought of among the babies characterized by irrational seizures, vomiting with drowsiness or lethargy, subdural hematomas, irritation, poor apatite, unresponsiveness, signs of physical abuse in case of acute injury. The babies having a sneaking suspicion of suffering from shaken baby syndrome are to undergo physical and neurological investigations along with radiological skeletal analysis, CT scanning of the skull. An opthalmologic consultation is also essential within the initial days of treatment. The detection of the shaken baby syndrome is confirmed with the existence of the three symptoms: the baby has the signs of head injury like subdural or subarachnoid hemorrhages, cerebral edema or skull ruptures; signs of retinal hemorrhages and the injuries are relevant to shaken baby syndrome and not any other consequences of injury. Initially there is reluctance to disclose the shaking incident by the parents or other caretakers which misleads the detection process. (Spaide; Swengel; Scharre; Mein, 1990)

Therefore the initial clinical demonstration of the SBS is not particular and may not instantly indicate the abuse. According to Caffey the SBS findings indicate the lack of any external injury to the head, face and neck simultaneously with aggressive intracranial and/or intraocular bleeding. More often the SBS is puzzled with meningitis, bleeding disorders, sepsis or really accidental injury. The baby victimized to the SBS may demonstrate a range of producing symbols. In cases of low intensity the signs incorporate vomiting, trend of poor feeding, lethargy or irritation, hypothermia, failure to thrive, increased sleeping and difficulty arousing, and failure to vocalize. In cases of acute intensity the SBS symptoms include signs of the fatal and irreversible damages. In extreme cases there is seizures and complete cardiovascular collapse.

The metaphyseal characteristics lead to conclude of a physical abuse when they are effected to at about the extremes of the bones. The traction lesions of the periosteum of the long bones without even ruptures at he ends, according to Caffey are considered to be an indication of physical abuse. Intensified retinal hemorrhage without other forms of ocular symptoms is taken to be a reinforced sign of the intracranial trauma occurred as a result of shaking. Even though severe accidental head injury is often combined with retinal hemorrhage yet are not normally so widespread. Wallis and Goodman lay emphasis on the group of injuries and the circumstances under which they are found out rather than stressing upon any single physical indication. (Smith, 2003) shaking injury is more confirmed by resorting to the diffusion weighted magnetic resonance imaging method which is considered to be most…

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References

Blumenthal, Ivan. (2002) "Shaken Baby Syndrome" Postgraduate Medical Journal. Volume: 78; No: 1; pp: 732-735. Retrieved at http://pmj.bmjjournals.com/cgi/content/abstract/78/926/732Accessed on 6 November, 2004

Burke, Sheila. (3 April, 2004) "Father found guilty in shaken baby syndrome case" Retrieved at http://tennessean.com/local/archives/04/04/49367567.shtml-Element_ID=49367567Accessed on 6 November, 2004

Folmer, Tiffany S; McCabe, Paul C. (September 2003) "Shaken Baby Syndrome: Implications for School Psychologists" NASP Communique. Volume: 32, No: 1

Retrieved at http://www.nasponline.org/publications/cq321shakenbaby.html. Accessed on 6 November, 2004
James, King, W; MacKay, Morag; Sirnick, Angela. (January 21, 2003) "Shaken baby syndrome in Canada: clinical characteristics and outcomes of hospital cases" the Canadian Shaken Baby Study Group. Canadian Medical Association Journal. Volume: 168; No: 2; pp: 155-159. Retrieved at http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=140423&rendertype=abstractAccessed on 6 November, 2004
Showers, Jacy. (March-April, 1992) "Shaken Baby Syndrome: The Problem and a Model for Prevention" Children Today. Retrieved at http://www.findarticles.com/p/articles/mi_m1053/is_n2_v21/ai_13561150Accessed on 6 November, 2004
Smith, Julie. (May/June 2003) "Shaken Baby Syndrome" Orthopedic Nursing. Vol: 22; No: 3; pp: 196-203. Retrieved at http://www.nursingcenter.com/prodev/ce_article.asp?tid=414057#14Accessed on 6 November, 2004
Spaide, Richard. F; Swengel, Richard M; Scharre, Douglas W; Mein, Calvin. E. (April, 1990) "Shaken baby syndrome" American Family Physician. Retrieved at http://www.findarticles.com/p/articles/mi_m3225/is_n4_v41/ai_9020723/pg_2Accessed on 6 November, 2004
Scheibner, Viera. (August 2001) "Shaken Baby Syndrome Diagnosis on Shaky Ground" Journal of the Australasian College of Nutritional & Environmental Medicine. Volume: 20, No. 2, pp: 5-8; 15. Retrieved at http://www.acnem.org/journal/20-2_august_2001/shaken_baby_syndrome.htm. Accessed on 6 November, 2004.
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