Disorders of the Veins and Arteries Pathophysiology of Chronic Venous Insufficiency and Deep Venous Thrombosis Chronic venous insufficiency (CVI) pathophysiology is either the obstruction or reflux of venous blood flow (Gujja, Wiley, & Krishnan, 2014). The perforating veins valve normally functions by preventing the reflux of blood from the deep veins into the superficial veins. CVI occurs when there is venous hypertension that is caused by incompetent valves. Venous blood will escape

Pathophysiology What's Gone Wrong? CVI is generally an indication of blood stasis or venous reflux, most commonly valvular incompetence in the low-pressure superficial venous system. The inability of blood to return to the heart from the legs causes it to pool and clot. CVI generally occurs within the deep veins (Deep Vein Thrombosis), may also be related to varicose twisting, valve malformations or pelvic tumors. Risk Factors Obesity, inactivity, pregnancy, smoking and extended periods

Chronic Venous Insufficiency and Deep Vein Thrombosis There are a number of conditions that affect the circulatory system in different ways. Not all circulatory issues are connected with blood clotting or high cholesterol. In fact, chronic venous insufficiency (CVI) is more associated with some form of trauma that damages the tissues of veins and reduces their ability to circulate blood effectively. This is much different to Deep Vein Thrombosis, which is

2004: 45). Recommendations Many recommend use of minimally invasive techniques including SEPS to treat and address problems related to chronic venous insufficiency (Kalra & Glovisczki, 2002). Multiple studies confirm the safety and efficacy of SEPS when used early, especially resulting from its low complication rates compared with other procedures including the formerly popular Linton procedures (Kalra & Gloiscki, 2002; Lee, et al. 2003; Tenbrook, et al., 2004; Bianchi, et al. 2003). More

Comparison of pathophysiology of CVI and DVT The pathogenesis of CVI is not completely understood; however, it's based on both venous reflux and obstruction; or an amalgamation of the two. Though venous reflux is actually based on a number of mechanisms, the key elements are venous valve ineffectiveness, vessel wall swelling, hemodynamic elements and additionally venous hypertension. These systems could be further exasperated by dysfunctional pumping devices (vascular and/or muscle pump),

Mostly proteins and platelets combine to form blood clotting in order to heal wounds inside the arteries. However, the accumulation of fatty protein and platelet deposits on the artery walls can sometimes cause the arteries to become narrow and hardened. This makes the artery more prone to damage and if it ruptures, the blood clotting occurs and thus, the artery becomes infected by arterial thrombosis. Impact of patient age on